Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the flip side, our ability to rescue SERT operate in KI midbrain synaptoneurosomes via the inhibition of FAK suggests elevated FAK signaling downstream from the Pro32Pro33 mutant, as verified by enhanced pFAK localization in five-HT https://walesj431nzk2.azuria-wiki.com/user