Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. On the other hand, our power to rescue SERT functionality in KI midbrain synaptoneurosomes via the inhibition of FAK implies elevated FAK signaling downstream on the Pro32Pro33 mutant, as confirmed by improved pFAK localization in 5-HT https://tonyy110qhy9.wikibyby.com/user